Dr. Robert J. Schloesser is the Associate Chief Medical Information Officer and Director of Point-of-Care Research & Innovation at Sheppard Pratt Health System; his extensive work focuses on bringing innovation from bench to bedside. Dr. Schloesser studied medicine at Ludwig-Maximilians-University, and completed his residency at the University of Maryland/Sheppard Pratt residency training program. Dr. Schloesser completed a postdoctoral fellowship at the National Institute of Mental Health’s Laboratory of Molecular Pathophysiology, and has also served as a guest lecturer at the Lieber Institute for Brain Development. In addition to his research and clinical work, he also currently serves as a clinical assistant professor of psychiatry at the University of Maryland School of Medicine. Dr. Schloesser is board certified in general psychiatry.
Ludwig-Maximilians University/Medical School Hannover
University of Maryland/Sheppard Pratt Residency Training Program
- Member, American Psychiatric Association
- Member, Society of Biological Psychiatry
- Member, Maryland Psychiatric Society
Hill JL, Hardy NF, Jimenez D V, Maynard KR, Kardian AS, Pollock CJ, Schloesser RJ, Martinowich K (2016): Loss of promoter IV-driven BDNF expression impacts oscillatory activity during sleep, sensory information processing and fear regulation. Transl Psychiatry. 6: e873.
Maynard KR, Hill JL, Calcaterra NE, Palko ME, Kardian A, Paredes D, Sukumar M, Adler BD, Jimenez D V, Schloesser RJ, Tessarollo L, Lu B, Martinowich K (2015): Functional Role of BDNF Production from Unique Promoters in Aggression and Serotonin Signaling. Neuropsychopharmacology. 1-13.
Schloesser RJ, Orvoen S, Jimenez D V, Hardy NF, Maynard KR, Sukumar M, Manji HK, Gardier AM, David DJ, Martinowich K (2015): Antidepressant-like Effects of Electroconvulsive Seizures Require Adult Neurogenesis in a Neuroendocrine Model of Depression. Brain Stimul. 8: 862-7.
Schloesser RJ, Jimenez D V, Hardy NF, Paredes D, Catlow BJ, Manji HK, McKay RD, Martinowich K (2014): Atrophy of pyramidal neurons and increased stress-induced glutamate levels in CA3 following chronic suppression of adult neurogenesis. Brain Struct Funct. 219: 1139-48.
Benninghoff J, Rauh W, Brantl V, Schloesser RJ, Jimenez D V, Moessner R, Möller H-J, Rujescu D (2013). Cholinergic impact on neuroplasticity drives muscarinic M1 receptor mediated differentiation into neurons. World J Biol Psychiatry. 14: 241-6.